| Customization: | Available |
|---|---|
| Powder: | Yes |
| Customized: | Non-Customized |
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GMP Factory Supply Lidocaine Powder Lidocaine Hydrochloride/Lidocaine HCl
Lidocaine hydrochloride is characterized by strong penetration, strong dispersion, rapidly onset. The anesthetic performance is twice that of procaine and the toxicity is1. There is an anesthetic effect after 5 minutes treatments, and anesthesia can last 1 to 1.5 hours, 50% longer than procaine. The drug is effective on the heart of the disease or arrhythmia caused by cardiac glycoside, but on the supraventricular tachycardia is poor. This product is fast and oral ineffective, with short duration, and often used as intravenous administration.
| Product Name | Lidocaine |
| Other names | Lignocaine, Gravocain, Maricaine, lignocainum hydrochloricum |
| CAS NO | 137-58-6 |
| Molecular Formula | C14H22N2O |
| Molecular Weight | 234.337 |
| Appearance | White crystalline powder |
| ITEMS | SPECIFICATIONS | RESULTS |
| Appearance | White crystalline powder | Pass |
| Purity | 99.00%-101.00% | 99.52% |
| Identification | Chloridate | Positive |
| Test A | Positive | |
| Melting point | 74-79 degree | 76-78 degree |
| Appearance of solution | Clear and colourless | Pass |
| Water | 5.5-7.0% | 6.7% |
| Acidity PH | 4.0-5.5 | 5.2 |
| Sulphated ash | 0.10% max | 0.08% |
| Heavy metals | 5ppm max | Pass |
| 2, 6-Dimethylaniline | 100ppm max | Pass |
Application
1.Lidocaine hcl is a local anesthetic and antiarrhythmic drug. It is clinically used for infiltration anesthesia, epidural anesthesia, surface anesthesia (including in the thoracoscopy or abdominal surgery for mucosal anesthesia) and nerve conduction block.
2.Lidocaine hcl is an amide local anesthetic. With the dose increased, the role or toxicity increased, there is an anti-convulsive effect with sub-poisoning plasma concentration; Blood concentration of more than 5μg • ml-1 can occur convulsions.
3.Lidocaine hcl in low doses can promote outflow of K+ in cardiomyocytes, reduce myocardial autonomy, and has antiarrhythmic effects. Increased plasma concentration may cause slowing of heart conduction, atrioventricular block, inhibition of myocardial contractility and decreased cardiac output.
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